Copper Peptide GHK
GHK-Cu
GHK-Cu (Glycyl-L-histidyl-L-lysine copper complex) is a naturally occurring copper-binding tripeptide found in human plasma. Preclinical research has investigated its role in stimulating collagen and glycosaminoglycan synthesis, promoting wound healing, modulating anti-inflammatory pathways, and activating tissue remodeling processes. It has been studied in dermal, bone, and nerve repair models.
Research Benefits
Key Research Applications
Documented areas of investigation based on peer-reviewed preclinical and clinical literature. For research reference only.
Accelerated Healing
Supports rapid tissue repair and wound closure mechanisms in preclinical models.
Cellular Regeneration
Promotes cellular migration and proliferation essential for tissue regeneration.
Anti-Inflammatory
Modulates inflammatory pathways to support the healing environment.
Structural Integrity
Supports collagen synthesis and extracellular matrix remodeling.
Literature
Research Highlights
Stimulation of collagen and elastin synthesis in human fibroblast cultures (Pickart et al.)
Accelerated wound closure and reduced scarring in rodent excisional wound models
Anti-inflammatory effects via TNF-α and IL-6 suppression in macrophage assays
Nerve regeneration and myelination support observed in peripheral nerve injury models
Specifications
Technical Data
Molecular Weight
340.38 g/mol (free peptide); 402.9 g/mol (Cu complex)
Sequence / Structure
Gly-His-Lys · Cu²⁺
Storage Conditions
Lyophilized: −20°C, stable 24 months. Protect from light. Reconstituted: 4°C, use within 14 days.
Solubility
Soluble in water at ≥10 mg/mL
HPLC Purity
Verified — Batch VRC-GHK-2406
Testing Lab
SGS Life Sciences · Jun 2026
Research Areas
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For Research Purposes Only. GHK-Cu is sold exclusively for in vitro laboratory research. Not for human consumption, diagnostic, therapeutic, or veterinary use. Research highlights are summaries of published preclinical and clinical studies and do not constitute medical advice or claims of efficacy. See our Research Use Disclaimer.